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1 Collectively, tumor-intrinsic mechanisms, spanning genetic mutation, epigenetic reprogramming, immune checkpoint activation, and cellular plasticity, converge to create a highly adaptable, treatment-refractory phenotype
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Any treatment plan should specify what will be monitored, when it will be reassessed and what symptoms require discontinuation or urgent care
In contrast, in co-formulation 38, comprising HP-B-CD (Average MS 1.08), was not physically stable after 14 days
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