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Modulation of reactive oxygen species (ROS) in pancreatic cancer: insight into tumor growth suppression by the superoxide dismutases
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Funding This study was supported by the National Key R&D Program of China (2022YFF0608401, 2022YFF0608404), the National Natural Science Foundation of China (92478101) and the Sichuan Science and Technology Program (2024NSFSC1145)
published in the Journal of Applied Physiology confirmed that BPC-157 significantly accelerated outgrowth of Achilles tendon explants, markedly increased in vitro tendon fibroblast migration in a dose-dependent manner, and substantially increased fibroblast survival under HO-induced oxidative stress establishing a three-part cellular mechanism (outgrowth, migration, survival) that distinguishes BPC-157s tendon repair profile from direct mitogenic agents
