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29 G 2 /M cell cycle phase arrest was observed within 24 hours of exposure to 5 mM DHA, with no recovery from G2/M arrest seen by 72 hours
4b, 5b), however notable conformational differences were observed around residues (acylated K1 Cagri , 14E Cagri , 17R Cagri and 37P Cagri ) that diverge from rAmy and pramlintide, an Amy peptide approved clinically for diabetes 6,7
The added glucagon receptor activity is a key research distinction, not a reason to self-select one treatment over another
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