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Reduced palmitoylation of SQSTM1/p62 in huntington disease is associated with impaired autophagy

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While passive accumulation via the enhanced permeability and retention effect can increase tumor drug levels modestly, active targeting through ligand-receptor interactions enables selective binding to cancer cell membranes, triggers receptor-mediated endocytosis and delivers intact nanoparticles directly into the cytoplasm, thereby bypassing P-glycoprotein efflux pumps stationed at the plasma membrane [120, 121]