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Taken together, the above-mentioned data suggest that GSH might serve as an important wound signal that triggers wound-induced systemic [Ca 2+ ] cyt transmission, activates JA biosynthesis, and regulates plant defense responses

Our data from PDAC cell lines showed that genetic ablation of xCT transporter leads to intracellular cysteine deficiency, and therefore GCN2-ATF4 pathway activation (16)

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