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J Androl 2012;33(2):170-175
In addition to these findings on atherosclerotic plaque formation, GLP-1R agonists prevented the increase in plasminogen activator inhibitor type-1 (PAI-1) and vascular cell adhesion molecule-1 (VCAM-1) gene expression in response to TNF- or hyperglycemia in HUVEC through PKA pathway [24],[70]
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Secondary BAss are particularly effective in triggering these responses because of their increased hydrophobicity and longer epithelial contact time
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