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At the same time, it was suggested that the mice treatment with glutathione (GSH) inhibited the animal deaths caused by X-ray irradiation [2, 3]

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Together, these results indicate that decreased GSH/GSSG redox/antioxidant capacity and increased oxidative stress in the autism brain may have functional consequence in terms of a chronic inflammatory response, increased mitochondrial superoxide production, and oxidative protein and DNA damage