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Conclusions Subclinical studies and epidemiological observational studies indicated that exposure to dioxins as persistent endocrine-disrupting chemicals is a cause of metabolic dysfunction associated with steatotic liver disease, as evidenced by hepatic steatosis and the presence of obesity, type 2 diabetes, and abnormal lipid profiles, which is due to the activation of hepatic aryl hydrocarbon receptor
Conversely, the treatment effect was more modest in patients with T2D compared to those without, indicating that GLP-1RA may provide additional clinical benefits through mechanisms beyond glycemic control
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