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In conditions such as Alzheimers disease (AD), Parkinsons disease (PD), multiple sclerosis (MS), amyotrophic lateral sclerosis (ALS), frontotemporal dementia (FTD), Huntingtons disease (HD), and Lewy body dementia (LBD), dysregulated glial activity, marked by sustained microglial and astrocytic activation, initiates a cascade of cytokine release, oxidative stress, and impaired neuronal support

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Pro-inflammatory cytokines like TNF-alpha, IL-1beta, and IL-6 damage surviving nerve fibers, interfere with Schwann cell repair functions, and create a hostile environment for axonal regrowth