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Hepatic taurine synthesis is largely restricted by the low availability of cysteinesulfinate as substrate for cysteinesulfinate decarboxylase, and taurine production is increased when cysteinesulfinate increases in response to an increase in the hepatic cysteine concentration and the associated increase in cysteine dioxygenase activity (Stipanuk 2004)
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Cell (2016) 167:45770:e13
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Understanding this mechanism matters because it explains why most GI side effects are dose-dependent, temporary, and manageable through titration strategies
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