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Bibcode:2017FrPS....8..346L

In the early stages of DKD, increased hyper-reabsorption and gluconeogenesis expose proximal tubular cells to hypoxia, resulting in mitochondrial dysfunction, increased oxidative stress and tubular injury ( Mitochondria is the primary organelle of in PTECs that is modulated to oxidative stress and hypoxic injury in the diabetic state ( Although there are a large number of in vitro and in vivo studies investigating the mechanism of hypoxia-induced renal tubular damage, due to the lack of non-invasive means, there are few clinical studies that have evaluated renal hypoxia in detail

The lack of multi-drug delivery platforms and long-term stability further restricts the clinical utility of PLGA nanosystems

Beginners may start with a single dose before bed (e.g., lower g), while advanced users may use multiple injections daily (morning and pre-bed) or increase the dose gradually