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Mechanistically, p53 induces ferroptosis by directly inhibiting SLC7A11 via system Xc(-) or by upregulating recombinant spermidine/spermine N1-acetyltransferase 1 (SAT1) downstream of p53, which is involved in polyamine metabolism (23)
FTO shapes appetite and fat-mass set-point, MC4R anchors the satiety circuitry, and TCF7L2 links to the glucose homeostasis that GLP-1 signaling influences
Nakagawa et al., 2005
Graefe's Arch Clin Exp Ophthalmol 2009, 247: 699706
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