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Thus, TB-500 may be hypothesized to support angiogenesis by combining a cytoskeleton-linked increase in endothelial motility with signaling changes that raise pro-angiogenic programs (VEGF-A and Ang2/Tie2) via Notch/NF-B coupling in damaged cellular structure
These results highlighted PTX-CSNPs as a viable nanotherapeutic option for conditions involving oxidative stress and inflammation, particularly in drug-induced nephrotoxicity
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