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Additionally, recent studies have reported that gstrodin and ginsenoside can improve neurological dysfunction and myocardial ischemia by inhibiting ferroptosis-related pathways (89, 90), suggesting that they may also exert therapeutic effects on osteoporosis through this mechanism, providing a potential therapeutic approach
Global DNA hypermethylation is independent of established risk factors and associated with an increased risk of insulin resistance 31
In contrast, ICV-STZ untreated rats showed a significant increase in the number of degenerated neurons and glial cells to approximately triple the value of control normal rats
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