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Fulminating hepatic failure and death are likely if the plasma levels are high

The xc/glutathione peroxidase 4 (GPX4) pathway, serving as the primary antioxidant mechanism defensing ferroptosis, is another widely investigated canonical pathway (191)

Several complex signal transduction pathways and cellular processes have been implicated in APAP hepatotoxicity, including those involving c-Jun N-terminal kinase, Nuclear factor E2-related factor 2 (Nrf2), p53, endoplasmic reticulum stress, autophagy, and sterile inflammation (Chowdhury et al

A functional genomic screen in vivo identifies CEACAM5 as a clinically relevant driver of breast cancer metastasis