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aureus impairs neutrophil function and produces anti-inflammatory metabolites ( Figure 4B ), initiating phagocytosis-induced cell death to suppress an overactive immune system
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357,358 In pyroptosis-mediated resistance, interventions targeting inflammasome components (e.g., caspase-1 inhibitors) or downstream fibroblast reprogramming (e.g., CCR6 antagonists) may limit fibrotic niche formation and CSC expansion
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