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This may leave p53 unbound, so that it may target senescent cells for apoptosis
This is in sharp contrast to GSH, which can enter cells only slowly via specific transporters
These conditions share common mechanisms involving disrupted redox homeostasis, elevated oxidative stress, and impaired detoxification, underscoring the therapeutic potential of strategies aimed at restoring glutathione balance to mitigate disease progression
J., Wu, M
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