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Interestingly, while the role of GSTs has classically been associated to the modification of small molecules and the role of glutaredoxins to the deglutathionylation of proteins, it has recently been shown that human GSTO1-1 can deglutathionylate -actin, which should prompt us to consider proteins as GST substrates (Menon and Board, 2013)
Nicotinamide N-methyltransferase as a potential therapeutic target in neurodegenerative diseases
10.3892/mmr.2018.8760 Summary Keywords cardiovascular disease (CVD), microRNAs (miRNAs), flavonoids, therapeutic modulation, cardioprotection Citation Prananda AT, Halim P and Syahputra RA (2025) Targeting miRNA with flavonoids: unlocking novel pathways in cardiovascular disease management
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