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GCL comprises a catalytic (GCLC) and a modifier (GCLM) subunit, and its activity can be readily inhibited with DL-buthionine-sulphoximine (BSO) [13]
Younes, R
The medication's dual agonism of GIP and GLP-1 receptors triggers physiological changes that may collectively influence cardiovascular parameters, though many proposed mechanisms are based on GLP-1 receptor agonist class effects and require further confirmation specific to tirzepatide
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