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3.3 Protein damage and the stress sensing Cells convert heat-induced protein damage into a regulated transcriptional signal through a competitive release mechanism in which the dissociation of HSF1 from inhibitory chaperone complexes serves as a central switch in the stress response (Figure 2a) (Zheng et al., 2016)

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Alterations in DNA damage repair (DDR) Molecular mechanisms in DDR DDR safeguards genomic stability under genotoxic stress, and its deficiency confers pronounced sensitivity to genotoxic therapies and PARP inhibitors while also driving resistance through repair restoration or pathway bypass

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