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Researchers who need to reason about dosing frequency in an experimental model generally rely on the exposure and dosing schedules reported in the specific animal papers rather than on a defined human-style half-life, and route decisions are governed by their study design
Complement activation, cytotoxicity, and alterations in cell junction proteins (e.g., reduced connexin-43) may also exacerbate tissue injury(Wroska et al., 2024)
Every direct comparison is cross-trial inference
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