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In the present study, dietary L-carnitine supplementation improved lipid catabolism significantly, but increased the whole body glycogen deposition in the fasting state, indicating that glucose degradation was inhibited
However, since iron plays crucial roles in redox regulation of normal cells, systemic administration of iron modulators may cause severe adverse effects (Tables 2,3)
Furthermore, C18:1-carnitine has been demonstrated to function as an efficacious noncompetitive inhibitor of GlyT2, which has the potential to serve as an analgesic by regulating the transport of glycine in the central nervous system (CNS) and influencing the concentration of this pivotal inhibitory neurotransmitter [112]
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