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Thomas, C
published in the Journal of Applied Physiology confirmed that BPC-157 significantly accelerated outgrowth of Achilles tendon explants, markedly increased in vitro tendon fibroblast migration in a dose-dependent manner, and substantially increased fibroblast survival under HO-induced oxidative stress establishing a three-part cellular mechanism (outgrowth, migration, survival) that distinguishes BPC-157s tendon repair profile from direct mitogenic agents
Saxena, A
Pathophysiological conditions such as inflammation and ageing [36] drive the cytosolic GSH/GSSG-coupled redox potential to be more positive (oxidising)
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