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One is experiments in mice in which induced overexpression of FGF21 as well as pharmaceutical administration of an FGF21 analogue both lead to reduced preference for alcohol in animals
Human fibroblast cells infected with herpes simplex-1 virus were shown to help support the suppression of virtual replication and helped replenish intracellular GSH levels in the presence of SAG
Another potential contributor is the medication's effect on the gastrointestinal system more broadly
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