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Ongoing investigations in our laboratory and clinical program aim to further substantiate this surprising observation in MDS and in clonal hematopoiesis of indeterminate significance (CHIP) and in clarifying the mechanism behind these effects. Further analysis of prospective cohorts with serial biospecimens would be advantageous to understand the potential connection between GLP-1 exposure and inflammasone markers and how this could influence ROS and DNA damage
Sigman M, Glass S, Campagnone J, Pryor JL
Urolithin A promotes mitophagy and suppresses NLRP3 inflammasome activation in lipopolysaccharide-induced BV2 microglial cells and MPTP-induced Parkinsons disease model
At 58 M and 580 M, cytotoxicity was not observed following 24 h treatment with all the samples
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