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Selective impairment of this carrier-mediated transport can contribute to selective vulnerability of tissues to specific toxins: for example, chronically ethanol-fed hepatocytes become selectively depleted of mitochondrial glutathione because of defective carrier-mediated transport, which makes the hepatocytes susceptible to damage by reactive oxygen species generated from the oxidative metabolism of ethanol (22)
Medina DA, et al
Further, it was speculated that improved mitochondrial function and reduced inflammatory processes by hexarelin contributed to this effect [6]
PMID 16434977
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