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coli, respectively, as described previously 20,48
It works by disrupting the interaction between the FOXO4 protein and p53, a key regulator of cell survival
Transcriptome analysis and results of qRT-PCR showed that most SlUGT genes could be induced by CHT, and the expression of these genes was regulated by glutathione
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removes ROS through superoxide dismutase activity, regulates the GPX4/SLC7a11 axis and Nrf2/HO-1 pathway to inhibit ferroptosis, forming a triple renal protective mechanism of ROS removal-ferroptosis inhibition-inflammation relief [77]
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