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It is more likely that endotoxins can either be absorbed directly through the enterocyte apical cell membrane or by a receptor-mediated transcytosis followed by exocytosis at the basolateral epithelial cell membrane [47, 49,50,51] (Fig
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BPC 157 activated the Src-Cav-1-eNOS signal pathway and reduced eNOS/Cav-1 binding in vascular endothelial cells It is known that phosphorylation of Src can promote Cav-1 activation and subsequent endocytosis of tyrosine kinase receptors and eNOS activation 20,24
Folia Microbiol
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