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Reviews of the field repeatedly note that the strongest evidence sits in animal and preclinical models , not in large, well-controlled human trials
Redox-controlled reversible modifications regulate UPR signaling, for example, thiol oxidation of ER molecular chaperones such as the binding immunoglobulin protein/78-kDa glucose-regulated protein (BiP/Grp78) that enhances the release of BiP from ER stress sensors and activates UPR (Wang et al., 2014)
High doses too frequently may lead to rare side effects like low potassium or vascular stress
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