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Co-agonism showed, on murine models, the capability to improve insulin secretion and sensitivity in a weight-dependent and weight-independent manner, probably through its action on nutrient metabolism (49, 50), preventing their accumulation and thus making these organs actively involved in metabolism with an improvement in insulin sensitivity and WL, yielding long-lasting effects
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40,41 Of note, Accelerated weight loss promotes cholesterol supersaturation of bile and impairs gallbladder contractility, facilitating gallstone formation, a pathophysiological process well recognised following bariatric surgery and during very low-calorie dietary interventions
