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VEGF is a potent pro-angiogenic agent that stimulates endothelial cell proliferation and migration
Thymosin beta-4, connective-tissue organisation and rat wound repair
A mechanistically curious reader is left to reconstruct the gastric story from receptor biology, pramlintide analogy, animal work, and side-effect patterns, rather than from a direct, cagrilintide-specific, longitudinal emptying dataset
Key documented mechanisms include collagen synthesis stimulation at nanomolar concentrations, modulation of matrix metalloproteinases and their inhibitors, fibroblast activation, angiogenesis promotion through copper-dependent endothelial signaling, and anti-inflammatory gene expression effects
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