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Previously, we have shown that chemogenetic activation of GIPR-expressing cells in either the hypothalamus or the hindbrain supresses food intake 7,8 , and GIPR-expression in oligodendrocytes has recently been linked to enhanced access of co-administered GLP-1R agonists 23
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In contrast, blood glucose level of 54 mg/dL or less occurred in 6% of patients receiving the 14 mg tablets, 3% of patients receiving placebo, and 0% of patients receiving the 7 mg tablets
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