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Co-agonism showed, on murine models, the capability to improve insulin secretion and sensitivity in a weight-dependent and weight-independent manner, probably through its action on nutrient metabolism (49, 50), preventing their accumulation and thus making these organs actively involved in metabolism with an improvement in insulin sensitivity and WL, yielding long-lasting effects
Genetic Predispositions and Metabolic Markers Influencing Non-GLP-1 Response Genetic variations in appetite-regulatory genes, dopamine signaling, and lipid metabolism pathways influence how individuals respond to non-GLP-1 agents
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