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There was no correlation between body weight and running time upon MOTS-c treatment (Supplementary Fig

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Renin, released from the juxtaglomerular cells, cleaves angiotensinogen to angiotensin I which is then cleaved to the bioactive form, angiotensin II, via the action of angiotensin converting enzyme (ACE)

The mechanism is progressive growth hormone-mediated lipolysis depletes triglyceride stores in visceral adipocytes over time, but the rate slows as adipocyte volume decreases