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These results were further supported by another study from the same lab, whereby GLP-1 was shown to stimulate myocardial glucose uptake via mechanisms that were p38 MAP kinase and NO dependent, but independent of adenylyl cyclase or Akt [73]
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doi: 10.3168/JDS.2016-11812 128 VanceJ
Although secondary apoptosis was thought to be an in vitro phenomenon for a long time, this effect was more recently observed in vivo
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