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Inactivation of the Cardiomyocyte Glucagon-Like Peptide-1 Receptor (GLP-1R) Unmasks Cardiomyocyte-Independent GLP-1R-Mediated Cardioprotection
Not every patient needs or benefits from the combination, which is why individualized assessment matters
These observations highlight why quality, not just quantity, of antibodies matter in determining pathogenicity, and suggest that polytherapy could be needed to curtail a variety of pathophysiological actions at the molecular level
The mechanisms underlying fatigue in patients taking tirzepatide are likely multifactorial
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