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GLP-1 medications are available in both injectable and oral forms, depending on the specific medication and its approved use

Access to GLP-1 therapy, the study suggests, often hinges on far more than a prescription

Importantly, both classes exhibit glucose-dependent mechanisms, meaning their effects diminish as blood glucose normalises, substantially reducing the risk of hypoglycaemia compared to sulphonylureas or insulin

A new Yale study shows that GLP-1 receptor agonists may help protect the liver during alcohol consumption while also raising blood alcohol levels in the process: bit.ly/4pCEOjE #Yale To view or add a comment, sign in Potassium levels and digoxin toxicity Mechanism: Digoxin inhibits Na /K + ATPase pump where the Intracellular sodium increase and indirectly increase calcium in the cells and helps in contraction ( positive inotropy) Vagal nerve stimulation= slower heart rate ( negative ) Hypokalemia : increase the digoxin toxicity ( less potassium is available for competitive binding ) Hyperkalemia: can occur because of digoxin toxicity ( Extracellular potassium increases ) Sign of toxicity Range : Normal: 3.5 - 5 mmol/L ( therapeutic range) mild hypokalemia: 3 -3.5 mmol/L : oral potassium supplements ( 20-40 mEq/ day in divided doses) moderate to severe : less than 3 mmol/L : 10- 20 mEq over 1 hour IV dilution with ecg monitoring maintain: 4 - 4.5 mmol/L in patients on digoxin Hyperkalemia : ( Insulin + glucose ) : shifts potassium into the cell