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Altogether, the results above provide evidence for a role of PDS5B in upregulating the DNA strand exchange activity of RAD51 and that its DNA binding attribute is indispensable for RAD51 enhancement

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Finally, a number of inherited mutations in genes involved in carnitine shuttling and fatty acid oxidation pathways do not systematically result in carnitine depletion (such that carnitine supplementation may not help mitigate the symptoms) but lead to abnormal profiles of acylcarnitine esters in blood (35, 40)