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Furthermore, our own work, together with that of others, emphasizes that a moderate elevation of circulating homocysteine related to impaired homocysteine metabolism (resulting from folate deficiency or dietary methionine excess) might trigger vascular disorders by promoting vascular spasm and thrombosis (Bellamy et al, 1998

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"Combination Therapies for Obesity: Cagrilintide and GLP-1 Receptor Agonist Synergy." Nature Reviews Endocrinology, 18(9), 541-556