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Exendin-4 lacked the amino acid alanine at one position in the polypeptide chain, making it more resistant to breakdown than human GLP-1
Their biological effects may be inappropriate for generalized metabolic disease but highly relevant for behaviorally mediated subtypes, reinforcing the need for stratified translational strategies
Plasma lipoproteins, liver function and glucose metabolism in haemodialysis patients: lack of effect of L-carnitine supplementation
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