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Post-cholecystectomy patients with certain predispositions may experience heightened gastrointestinal responses to GLP-1 activation due to the combination of genetic receptor patterns and altered bile physiology

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It upregulates the expression of anti-inflammatory genes, including NF-BIA , MAP3K8 , and TLR5 , while concurrently downregulating pro-inflammatory genes such as TNFSF6 , TNF- , and nuclear factor-B ( NF-B ) [36,37,38,39]