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Chronic stress and insulin resistance-related indices of cardiovascular death risk, part I: neurophysiological responses and pathological sequelae
Reuptake and Metabolic Inhibition : Ephedrine prolongs the residency of norepinephrine in the synaptic cleft by weakly inhibiting NET-mediated reuptake and demonstrating a weak inhibitory effect on mitochondrial monoamine oxidase (MAO), the primary enzyme responsible for catecholamine degradation (Statler et al, 2023)
PMID: 16901595
Rather than stimulating metabolism directly, it alters upstream behavioral drivers of energy consumption
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