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10.3390/molecules24112062 Molecules
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Human studies of glucagon receptor antagonism show a striking result: hepatic amino-acid clearance drops hyperaminoacidaemia develops skeletal muscle becomes the fallback source of amino acids This pattern mirrors the very changes we see under GLP-1RA therapy
Takeaway: When appetite is suppressed by GLP-1 medications, hitting protein targets requires deliberate planning and often supplementation
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