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Ganetespib acts as a radiosensitizer in pancreatic ductal adenocarcinoma by disrupting HIF-1, STAT3, and AKT signaling and inducing G2-M cell cycle arrest, thereby impairing DNA damage repair during irradiation

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To address this knowledge gap, we have developed a series of genetic models that permit spatiotemporal control over the expression of GCLC, the rate-limiting enzyme in GSH synthesis

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