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Like insulin, sulfonylureas also improve both -cell glucose sensitivity and GIP sensitivity by ameliorating hyperglycemia in patients with poorly controlled type 2 diabetes
This review examines the molecular mechanisms underlying ASM-associated oxidative stress in clinical, non-kindled/kindled preclinical, as well as non-epilepsy models, and highlights evidence demonstrating the ability of ascorbic acid to attenuate ROS accumulation, enhance endogenous antioxidant defenses, and modulate redox-sensitive signaling pathways implicated in epileptogenesis and ASM-induced toxicity
PMID: 2425690
