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Taken together, our data indicate that the loss of GSH triggered by Gclc deletion induces a defective mitochondrial accumulation in FoB, which may lead to compensation through upregulation of glycolytic metabolism
Intralipid therapy has moderate level evidence but lacks consistent large RCT confirmation and is not universally endorsed due to limited study quality
M.H.A.M
Myocardial Bioenergetics The potential to therapeutically augment cardiac function and reduce morbidity and mortality in HF by specifically altering myocardial metabolism has tremendous appeal and growing support from the scientific literature
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