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One hypothesis proposes that muscle insulin resistance results from decreased mitochondrial fatty acids oxidation
However, current evidence is still inconclusive to demonstrate a direct negative effect of GLP-1 RAs on LBM loss, largely due to heterogeneity in study designs and the limited number of trials specifically evaluating this endpoint [77]
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These findings highlight substantial heterogeneity in SUD comorbidity patterns, with alcoholnicotine related-disorders representing the most prevalent pairing in this population ( Supplementary Figure 1 )
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