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Considering the general knowledge about compensatory and adaptive changes that occur in the heart to preserve cardiac output, supporting studies demonstrated that GLP-1R signaling activation by agonism can activate also different close targets inducing either adverse drug reactions or beneficial actions through different receptor-mediated signaling pathways [61]
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The hypothalamus represents a primary site of GLP-1 action, particularly in areas regulating energy homeostasis and appetite
Nitrotyrosine proteome survey in asthma identifies oxidative mechanism of catalase inactivation
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