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These GIs are involved in meningococcal adhesion and subsequent host cell invasion, iron uptake ( hpuAB ), meningococcal survival in the intracellular environment, modulation of the host cell cycle, innate immune escape and evasion of phagocytic killing, epigenetic control of gene expression ( modB ), and competition within the meningococcal population in the human nasopharynx
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AGA clinical practice guidelines on the gastrointestinal evaluation of iron deficiency anemia
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