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This dual anti-inflammatory and pro-survival signaling complements the gene-level anti-inflammatory effects of GHK-Cu and the vascular protection afforded by BPC-157's nitric oxide production, creating coordinated suppression of destructive inflammation while preserving protective immune responses essential for optimal tissue repair

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Figure 3: Changes in the number of viable cells in the BPC 157-treated tendon fibroblasts following the addition of growth hormone Figure 4: Changes in PCNA gene expression in the BPC 157-treated tendon fibroblasts following the addition of growth hormone 3) The results of the study conducted by the research team of Pervoic et